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In agreement with observations made in in vitro models, various findings support the hypothesis that the cardioprotective effects of GLP-1 (736) in ischemia-reperfusion models are mediated by the activation of kinases in the RISK pathway, including PI3K, ERK1/2, cAMP, PKA, Akt, and P70S6K (Table 2).29,34,42,49,50,5255 On the other hand, it has been suggested that the improvement seen after the administration of GLP-1 (736) in MI in terms of increased survival and improved cardiac function could also be due to the decreased activation of inflammatory cells,56 improved myocardial microcirculation,57 and increased myocardial glucose uptake (Fig
