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The induction of membrane damage and the generation of reactive oxygen species resulted in cell lysis (Shilling et al

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Human safety data is insufficient to make any definitive statement about long-term use, with no large-scale toxicology or long-term safety trials in humans performed as of this writing

The increase in the bile acid pool may then stimulate TGR5 bile acid receptors on the L cell [62, 67], causing an increase in GLP-1 secretion via mitochondrial oxidative phosphorylation and calcium influx [67]