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In tendon transection model control animals had two peaks of FVIII positivity, announced by the higher number of CD34 and VEGF positive elements on the seventh day, in comparison to the BPC 157 animals

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These adverse effects stem from the entry of the cytoplasmic exonuclease TREX1 into the nucleus, which induces DNA cleavage [195], as well as the translocation of DNA repair factors such as Ku70, Ku80, and BRCA1 to the cytoplasm [190, 194]