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Therefore, a possible mechanism proposed by the authors is that the activation of the farnesoid X receptor (FXR) by exendin-4 stimulates the expression of FGF15 in the small intestine and inhibits the expression of cytochrome P450 family 7 subfamily A member 1 (Cyp7a1) in the liver through fibroblast growth factor receptor 4 (FGFR4), thereby reducing fecal bile acid excretion and liver bile acid synthesis [75]

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The development of NAFLD is initiated via the accumulation of excess fat within hepatocytes, a state known as simple steatosis [8]

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Benowitz AB, Scott-Stevens PT, Harling JD

10.7150/thno.34674 12 JiL.JiangP.LuB.ShengY.WangX.WangZ