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AHK-Cu works through stimulation of dermal papilla cells, reduction of TGF-beta-1, upregulation of VEGF (which builds new vessels rather than widening existing ones), and inhibition of apoptosis

Although redressing the BA composition has proved to be beneficial in mouse models, it was unlikely to translate to human pathophysiology, as changes in BA composition in mice hardly reflect similar changes in BA profiles in humans due to the differences in BA composition 41

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The safety profile of GHK-Cu is well-established given its endogenous origin

doi: 10.1093/aje/kwx246 Summary Keywords glucagon-like peptide-1 receptor agonists, erectile dysfunction, type 2 diabetes, obesity, Mendelian randomization Citation An H, Xie K and Gan H (2024) Glucagon-like peptide-1 receptor agonists and the risk of erectile dysfunction: a drug target Mendelian randomization study