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Moreover, when mice were pre-treated with vehicle or L2-Cmu (20 mg/kg) by intraperitoneal (i.p.) injection, and then challenged with an intravenous (i.v.) bolus of saline or 5 g IGF-1, L2-Cmu was able to modulate IGF-1R activation in vivo, as demonstrated by a near complete inhibition of IGF-1R phosphorylation in lung and ~70% inhibition in heart (Fig

Differential roles of M1 and M2 microglia in neurodegenerative diseases

Fogacci, F., Giovannini, M., Tocci, G., Imbalzano, E., Borghi, C., & G Cicero, A

aeruginosa increases with age for CF and PCD, people with NCFB are more likely to acquire and maintain chronic infection with P

No body weight loss was observed in the present study