It is resistant and stable in the human gastric juice (more than 24 h [42]), acting as a membrane stabilizer, counteracting leaky gut syndrome, as a particular target [19] distinctive from the standard peptide growth factors [16] (i.e., standard peptide growth factors are rapidly destroyed in human gastric juice [42]), with particular molecular pathways involved [15,19,20,28,43,44,45,46,47,48,49,50]
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FLNA splice variant-1 showed an increased binding to integrins with respect to wild-type non-spliced FLNA, suggesting that this alternative splicing might function as a regulatory mechanism for the binding of partner proteins to FLNA, with possible implications in pituitary tumors responsiveness to SS/DA analogs
& Smith, C
