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The mechanism is real: GHK-Cu activates Wnt/beta-catenin signaling, reduces androgen receptor sensitivity, and suppresses the perifollicular inflammation that quietly chokes follicles over time

All these proteins could enable HS plants to maintain their growth during the hot summer periods, particularly as their biomass was not as high as that of the plants at the cooler sites (MacAlister et al

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This suggests AHK-Cu primarily protects existing DPCs from death rather than simply making them multiply faster an important distinction for treating hair loss where follicle miniaturisation involves progressive cell loss

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