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ALS is typically a late onset disease with a mean age of onset at 64 years old (Marin et al., 2016)

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Notably, p53 plays a dual role in oxidative stress: in mild and moderate oxidative stress, it promotes the repair of genetic damage and cell survival by mediating cell cycle arrest and DNA repair

Mechanism: Binds to the Ghrelin receptor (GHS-R1a)

The first option has for instance been explored by targeting B cells in gMG, RA (NCT00931086), ITP, CIDP, Sjgrens syndrome (NCT03154385, NCT02531538, NCT03905525), and TED (NCT00212726) [383, 384] with therapeutic mAbs directed against CD20, B-lymphocyte stimulator protein, and CD40