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Researchers analyze how these mechanisms may contribute to maintaining tissue integrity in experimental environments, without concluding therapeutic effects

Considering the critical roles of these two metals in liver injury [Table 2], the mechanistic processes known as cuproptosis (copper-induced cell death) and ferroptosis (iron-induced cell death) merit intensive discussion [Figure 1]

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[37] have recently demonstrated that the N-terminally located glycine (Gly), present in the BPC 157 pentedecapeptide, can serve as a stabilizer that can regulate the compounds protease degradation [37]