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These are attributable to Cori and Krebs cycle disruptions, energy metabolism and mitochondrial function deregulations, decreased -hydroxybutyrate and acetate, and increased acetoacetate and acetone production, suggesting the point of low-level AFB 1 -induced -oxidation of fatty acids and suppressed tricarboxylic acid cycle (91)

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it's driven by hydrolysis and oxidation, both of which accelerate exponentially with temperature

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