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In vitro models of AD, PD and MS have demonstrated that SFN is efficient in counteracting cell death, neuroinflammation, oxidative stress and protein-aggregation induced by various toxins such as A plaques, LPS, 6-OHDA or hydrogen peroxide, through upregulation of the NRF2/ARE-axis (Table 2)

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(2019) Glutathione metabolism in renal cell carcinoma progression and implications for therapies, Int

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Interactions between biosynthesis, compartmentation and transport in the control of glutathione homeostasis and signalling

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