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Strategies to modulate immune cell infiltration and ROS production could potentially mitigate oxidative stress and limit tissue damage in IBD patients

It can also cause suppressed immune function

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TAK1 phosphorylates and activates the IB kinase (IKK) complex and MAPK kinases (MKKs), which in turn stimulates nuclear factor kappa B (NF-B) and activator protein 1 (AP-1) transcriptional activity leading to inflammation and cell survival (Mihaly et al., 2014)

although none activated G6PD better than AG1, several displayed AG1-like activation and are better suited for in vivo studies they lack a disulfide bond [103]

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