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593,599 Additionally, supplementation with NMN restores the activity of kidney SIRT1, thereby reducing the stress response by regulating the JNK pathway and protecting mice from cisplatin-induced AKI

In the advancement of treatment strategies, although approaches like mitochondria-targeted antioxidants (e.g., MitoQ) and macrophage metabolic reprogramming (e.g., SDH inhibitors) have shown significant efficacy in animal models, their clinical translation faces severe challenges in targeted delivery and microenvironment adaptation

New recent evidence might, therefore, indicate that erythrocytes are not just mechanistically disintegrated entities but might be capable of adjusting their cell fate based on incoming life/death signals

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Baytshtok, V