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Cartalax operates via the "Sequence-Specific DNA Binding Model." Its nanoscale size allow it to pass through nuclear pores and bind to the major groove of DNA

DN30 is involved in decreasing the c-Met signaling pathway through a variety of ways, including c-Met destruction, reducing c-Met expression, c-Met proteolytic detachment, and shedding c-Met from the cell surface, providing traps for HGF [162, 170]