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Materials and methods The meta-analysis was conducted per instructions outlined in the Cochrane Handbook for Systematic Reviews of Interventions (Reference Higgins, Thomas and Chandler13) and the Grading of Recommendations Assessment, Development, and Evaluation Handbook (Reference Schunemann14)

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Our major insight from examining the vitamin B 12 GSH data in conjunction with the Reed-Nijhout model is that cysteine-block explains why GSH does not increase as a result of vitamin B 12 deficiency induced hyperhomocysteinemia

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Alterations in both pathways enhance oxidative stress and increase APAP toxicity