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50,51 Moreover, GIP infusion significantly reduced CTX-I levels in healthy humans, 52 as well as in T1D or T2D, 53 indicating a robust suppression of bone resorption in various settings that is independent of the established effects of GIP on insulin secretion

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These findings highlight the potential of the ApoE-Ch mutation to protect against ApoE4-driven AD pathologies, offering a promising target for therapeutic development against AD

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