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Dulaglutide exploits this property of the Fc fragment to extend the half-life by linking the amino acid-substituted GLP-1 molecule to the N-terminal end of the Fc fragment via a 16-amino acid junction (Figure 2)

Synergistic interactions of N-3 PUFAs with multiple nuclear receptors, in particular PPAR- and RXR, might amplify VDR signaling further, as N-3 PUFAs promote PPAR-/PGC-1 pathways 115,116 and stabilize VDR-RXR heterodimers, which are critical for the VDR transcriptional activity 117,118