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The data presented here provide evidence in favour of altered mitochondrial metabolism and suppression of anabolism being major consequences of toxic doses of APAP in HepG2 cells, and that resulting lactic acidosis may be contributing specifically to cell death

Be skeptical of specific human half-life or bioavailability figures circulating online (see FAQ)

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Endocan serum levels in patients with non-alcoholic fatty liver disease with or without type 2 diabetes mellitus: a pilot study

Is there a difference between a 30mg vial and three 10mg vials