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ROS can be produced exogenously by ionising radiation (204), as well as xenobiotics, which can be pharmaceutical or environmental chemicals (205, 206)

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The absence of a drug affinity complex (DAC) yields a shorter functional half-life in experimental models (approximately 30 minutes to two hours [1]) allowing researchers to examine time-dependent signaling dynamics and pulsatile secretion patterns that more closely mirror endogenous GHRH activity