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Published animal studies using BPC-157, including those extending across multiple weeks of daily administration, have not documented tumor formation or accelerated malignant growth as an observed outcome

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During postprandial periods there is an increase in free circulating IGF-1 via insulin-induced suppression of IGFBP-1 secretion [168170], which sequesters free IGF-1 making it unavailable

The preclinical signal for BPC-157 is unusually consistent across independent research groups and across a wide range of injury models, pointing toward a compound with genuine biological activity rather than a laboratory artifact

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