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Authors affiliated with Broad Institute of MIT and Harvard, including corresponding author Stuart L

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Illustratively, L-NAME appears to be more sensitive to BPC 157 application, providing that BPC 157 nullifies the effect of L-NAME and brings all isoprenaline (+L-NAME) lesions markedly below control values (L-NAME + BPC 157 + isoprenaline

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A recent analysis using Mendelian randomization showed that genetically predicted homocysteine levels increased the odds of NAFLD.[ref] Related article: NAFLD genes Cardiovascular disease: To answer the question of whether a modestly elevated homocysteine level is causally associated with an increased risk of cardiovascular disease, researchers again used huge genetic data sets and Mendelian randomization to determine causality

doi:10.1093/clinchem/30.6.884