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Notably, brain-resident astrocytes and microglia might also be susceptible to EBV infection, which would greatly broaden the potential source of deleterious viral products and downstream effects, including reactivation of retroviral elements such as HERV-W (Hassani et al., 2018)
The results indicated that the oxidative stress and ROS levels induced by LPS and 2,3-cGAMP could be significantly inhibited by C-176 or GLP-1RAs
These enzymes may play critical roles in detoxifying reactive oxygen species, thereby protecting the cell from oxidative damage
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