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Early oncology nanomedicines were largely designed around exposure modification, including prolonged circulation, reduced peak-related toxicity, altered tissue distribution and partial exploitation of the enhanced permeability and retention effect [18, 20, 21, 33]

Consequently, there is a necessity to investigate the effect of glutamine metabolism and NK cell interaction on the proliferative effects of cancer cells in a more in-depth and broader systematic manner through multidimensional modeling and the integrated use of multiple biological approaches

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