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Evidence-based complement altern med: eCAM:5578574

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Therefore, targeting the Nrf2/ARE pathway represents a promising strategy to simultaneously address oxidative stress-induced neuronal dysfunction and the toxic proteinopathies characteristic of these disorders, offering hope for more effective treatments that modify disease progression rather than merely alleviating symptoms (Yang et al., 2025

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human GLP-1RA based analogues), the receptor activation is different between short-acting (on/off) and long-acting (continuous) compounds