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However, so far this has only been studied in vitro with SK-N-MC and Neuro2A cells (Sidharthan et al., 2013)

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Though these are a normal part of human metabolism, they cause significant oxidative stress over time, damaging DNA inside cells and leading to premature aging

Such biochemical changes are direct or indirect effects of the mutation and primary cell loss, but could potentially contribute to the development of secondary neuropathologies and functional changes in the brain of Lurcher mice

Cheng, J