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An enhanced potency is due to the markedly decreased binding of human Long-R3-IGF-I to IGF binding proteins which normally inhibit the biological actions of IGFs

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Previous studies established that CAT inhibits ferroptosis by activating transient receptor potential vanilloid type 1 (TRPV1), thereby upregulating GPX4 expression and ameliorating intestinal ischemiareperfusion injury as well as organoid hypoxia-reoxygenation injury (Deng et al., 2021)

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Mechanism: cyclooxygenase pathway cyclooxygenase pathway NSAIDs exert their effects, primarily, through the inhibition of cyclooxygenase (COX)-induced conversion of arachidonic acid to prostaglandins and thromboxane

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