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The APOE-TR model is a mouse knock-in model where exons 24 (comprising the mouse APOE gene) are replaced with human APOE in 129xC57BL/6 mice back-crossed to C57BL/6 mice (Sullivan et al., 1997)

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Recurrent MET fusion genes represent a drug target in pediatric glioblastoma

BT caused lipid peroxidation in NSCLC by accelerating the ubiquitination-dependent degradation of GPX4 and increasing the intracellular Fe 2+ level (Zhang W

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