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Study Design Highlights Study type: In vitro Caco-2 human intestinal epithelial cell model Conditions: Cellular stress model evaluating barrier function Outcomes: Intestinal barrier markers, tight junction proteins, intracellular ATP levels, beneficial bacteria growth and adhesion Key Findings (What the Study Showed) Improvements in markers related to intestinal barrier integrity including measures of cellular cohesion and permeability Supported expression of proteins involved in maintaining connections between intestinal cells (tight junction proteins) Increased intracellular energy (ATP) levels, suggesting support for normal cellular activity and function Enhanced growth and adhesion of beneficial bacteria in the intestinal cell model, indicating support for a healthy gut environment and microbehost interactions Benefits of AstraGin Demonstrated by This Study Based directly on the in vitro findings, AstraGin was shown to: Support intestinal barrier function markers under cellular stress conditions Promote tight junction protein expression supporting cellular structure Support intracellular ATP (cellular energy) levels Encourage beneficial bacteria growth and adhesion in gut cell models Why This Study Matters This cell model study provides mechanistic insight into how AstraGin supports intestinal health at the cellular level helping maintain normal barrier function, supporting cellular energy, and promoting a favorable environment for beneficial bacteria through complementary cellular pathways.* Reference Link: Study 5 Summary Complete Study Information: Astragaloside II Supports Cellular Activity, Amino Acid Uptake, and Intestinal Structure Scientific Reports

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Additional samples that have been mixed with EDTA are used for measurement of routine haematological analytes, such as haematocrit, erythrocyte concentration and haemoglobin concentration

Our data suggest that thyroid hormones modulate GH release induced by GHRH and GHRP-6 through different mechanisms

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