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The secretion of the toxin, CagA, by Helicobacter pylori , attenuates adhesions between adjacent IECs as well as reducing apical-basal cell polarity by altering the IEC cytoskeleton and encouraging TJ disassembly (Bagnoli et al

[1] The forward pathway is present in several bacteria, such as Escherichia coli [2] and Bacillus subtilis , [3] and involves the transfer of the thiol group from cysteine to homocysteine (methionine precursor with the S-methyl group), thanks to the -replacement of the acetyl or succinyl group of a homoserine with cysteine via its thiol group to form cystathionine (catalysed by cystathionine -synthase, which is encoded by metB in E

Yin, N., et al