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Although excess Mn can activate microglia and promote neuroinflammation (Tjalkens et al., 2017), astrocytes may be the more dominant mediator of Mn neurotoxicity in the brain

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10.1006/nimg.2002.1260 Neuroimage 73 LiB

Additionally, the peptide inhibits acetyl-CoA carboxylase activity in hepatocytes and adipocytes, the rate-limiting enzyme in fatty acid synthesis

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[2] The result is vasoconstriction and increased systemic vascular resistance, which can be therapeutically useful in conditions like vasoplegic shock where pathological vasodilation has caused life-threatening hypotension