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Since aging is a primary risk factor in neurodegenerative diseases, it is likely that the age-dependent reduction in microbial species diversity, including alterations in relative abundances of region-specific microbes, further facilitates persistent degradation and damage of gut mucosal and gut epithelial layers and increases infiltrations of bacterial toxins (Qin et al., 2007

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Endogenously, free radicals are by-products of normal cellular metabolism (Fig

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Here are several key factors contributing to this phenomenon: 1

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