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These data support that selectivity based on target-cell antigen density can be tuned through changes in monovalent affinity and avidity, which may reduce on-target, off-tissue toxicities

In cell studies, luteolin i nhibits TNF-alpha and IL-6 released via suppressing NF-B .[ref] TNF-alpha and IL-6 are linked to many chronic diseases caused by elevated inflammatory cytokines

Disrupting G6PD-mediated redox homeostasis enhances chemosensitivity in colorectal cancer