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Mechanistically, upon activation, CRTC1 interacts with the CREB transcription factor and enhances the expression of CREB target genes like LYPD3 , a high-glycosylated cell surface protein, 513 ultimately augmenting cell migration and invasion, which contributes to esophageal cancer progression

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The reaction of o-quinones with the thiol group of Keap1 cysteines is an essential mechanism for the activation of the NRF2/ARE axis, regulating and ensuring the appropriateness of redox responses and oxidative signaling factors [165], (Figure 11)