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Increased constitutive c-Jun N-terminal kinase signaling in mice lacking glutathione S-transferase Pi

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HEVs express high levels of addressins, such as PNAd and MAdCAM-1, which bind L-Selectin/CD62L on lymphocytes and support the migration of nave lymphocytes from the bloodstream into lymph nodes (128, 129)

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In conclusion, our study revealed a novel mechanism of neuronal injury induced by chronic hypoxia, highlighted the role of -syn phosphorylation in this process, and provided a potential new target for the intervention of chronic hypoxia injury