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GHK-Cu works through copper-dependent enzyme activity, extracellular matrix signaling, integrin pathways, MAPK/ERK cascades, and direct gene expression modulation

It's all so very complicated that you would probably benefit from seeing your GP, who should try and to sort this out for you

Multi-pathway engagement (ECM, oxidative stress, inflammatory tone)

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Peter also dives into the potential upside of GLP-1s beyond weight loss and what evidence would justify broader use, explains why transcranial magnetic stimulation is on his radar, and shares his framework for evaluating body fat percentage

Theoretically, they operate through different mechanisms: semaglutide suppresses appetite and slows gastric emptying via GLP-1 receptors, while AOD 9604 acts on beta-3 adrenergic receptors on fat cells