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4.1 Ferroptosis as an amplifier of inflammation Figure 4 presents a detailed mechanistic model of the ferroptosis-immune axis, illustrating the self-perpetuating cycle that is posited to be a core driver of COPD pathology

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In the OVA-induced asthma lung infiltrations cells, significant alterations to miRNA levels were evident, specifically miR-34a downregulation which targets FOXP3, and hence T regulatory functions [194]