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A novel HDAC/MIF dual-target inhibitor (6a) at a concentration of 12.5 M promotes apoptosis by inhibiting HDAC activity and inducing histone acetylation (H3K27ac), while simultaneously blocking the MIF-CXCR7-AKT signaling pathway, thereby synergistically inhibiting the survival and proliferation of NSCLC cells (especially EGFR-mutant/TKI-resistant strains)

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Kaidery NA, Banerjee R, Yang L, Smirnova NA, Hushpulian DM, Liby KT et al (2013) Targeting Nrf2-mediated gene transcription by extremely potent synthetic triterpenoids attenuate dopaminergic neurotoxicity in the MPTP mouse model of Parkinsons disease

Freezing reconstituted solutions is generally discouraged

The authors are presently preparing a review of the use of quercetin as cancer therapy