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However, time-varying factors, such as age, may still influence the association

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Moreover, in comparison to normal esophageal epithelium, ZNF282 has been frequently reported to be overexpressed in ESCC tissues and ZNF282 depletion increased apoptosis and promoted cell cycle arrest at G1/S, suggesting that ZNF282 transcriptional co-activator, plays pivotal role in controlling E2F1-mediated ESCC progression