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However, under pathological conditions, excessively activated NSPs become major drivers of tissue destruction, contributing to the development and progression of various inflammatory lung diseases ( The three primary NSPs exhibit both shared and distinct functional characteristics

Sabio, G

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identified a crucial role for the TRPV4-AMPK-NF-B pathway (where NF-B, which normally induces apoptosis, is phosphorylated via the AMPK hypothermia-regulating pathway) [129]