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Y., & Guo, X

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mutation of residues within the predicted receptor-cholesterol binding sites caused changes in basal receptor activity, ligand binding, G protein coupling and cAMP responses (21)

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Platelets can contact LSECs to induce LSEC proliferation and secrete VEGF and IL-6 via sphingosine 1-phosphate (S1P)

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MVs identified in the GHSR gene

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