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References McEwen BS, et al

Side effect incidence by dose The pattern is clear

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In particular, accumulating evidence suggests that inflammation is a prominent etiology of OA, and that chondrocytes and synovial cells overproduce many of the soluble inflammatory mediators, such as cytokines, chemokines, growth factors, adipokines, prostaglandins, leukotrienes and regulators such as CPB [7, 8]

For the initiation phase, where you need 1-2mg, reconstituting at 5mg/mL or 10mg/mL is far more accurate

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