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Notoriously, hyperhomocysteinemia-induced brain injury and disruption of BBB are mediated by the release of pro-inflammatory cytokine and immunomodulatory dysfunction to protect the injured brain (Zhang et al
CYP24A1, a gene that provides instructions for making the enzyme 24-hydroxylase, is involved in the catabolism of both 25(OH)VD 3 and 1,25(OH) 2 D 3, thereby limiting vitamin D receptor (VDR)/1,25(OH) 2 D 3 signaling 14
PMID: 28781108 (This report is OPEN ACCESS if you would like to read it) In the study, the investigators recruited 62 people with Parkinsons (average time since diagnosis was approximately 6 years) and they randomly assigned them to one of two groups, exenatide (the Bydureon formulation which is injected once per week) or placebo (32 and 30 people, respectively)
Kaemmerer et
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