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GLP-1 and GIP both participate in incretin biology, while glucagon receptor activation may influence energy expenditure, fat oxidation, and liver metabolism

European Neurology, 23 (5), 358-363

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GLP-1 analogs, such as Liraglutide and Exendin-4, unlike endogenously produced GLP-1, are not rapidly degraded by Dipeptidyl peptidase-4 (DPP-4) and, therefore, can induce sustained therapeutic actions, that otherwise would not be possible due to the exceedingly short half-life of endogenous GLP-1 in circulation