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Not only tirzepatide displayed a 20-fold lower potency in cAMP generation compared with native GLP-1, but also showed a signaling bias towards cAMP instead of beta-arrestin recruitment [39, 42]

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Unlike PDE-5 inhibitors, it acts directly on the central nervous system via melanocortin pathways

LL-37 forms complexes with self-DNA and self-RNA released from damaged skin cells

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