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This priming-dependent enhanced phosphorylation aligns with our in vivo post-translational modification analysis: S114 phosphorylation is consistently detectable, whereas downstream residues S117 and S120 remain elusive, likely due to the transient nature of their phosphorylation following S114 priming

[27] He then spent three years on fruitless efforts to persuade the pharmaceutical industry to develop exendin-4 into a drug

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No serious adverse events, hospitalization, organ damage, death, have been documented in any published BPC-157 research