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However, the GSAAT(C168S) and GSAAT(C190S) variants are hardly stimulated at all by additional reducing power

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91 In UC patients, endoplasmic reticulum function may be impaired, leading to abnormal accumulation of intracellular proteins and exacerbation of the cellular inflammatory response

This does not mean that GSH cannot be broken down

Statistical analysis We obtained the Wald estimate (genetic association with CVD and its risk factors divided by the genetic association with l-carnitine) for each SNP