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Doxorubicin induces endotheliotoxicity and mitochondrial dysfunction via ros/enos/no pathway

Another potential regulator of the chronic GSH adaptive response to CS exposure are the glutathione-S-transferases (GSTs) that conjugate GSH to xenobiotics for transport that could stimulate an increase in GSH synthesis to match GSH transport

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Documented in animal-model literature for tissue remodeling, GI mucosal barrier signaling, and the cytoprotection paradigm

Front Cardiovasc Med 8:642181

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