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It is not intended for use as a drug, food, cosmetic, or medical product, and must not be used for human or veterinary administration

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These neurophysiological shifts were observed in preschoolers with ASD (35 males and 11 females) diagnosed through to DSM-5 and validated with ADOS-2 and ADI-R, with participants screened to exclude neurological syndromes, epilepsy, sensory impairments, gastrointestinal diseases, secial diets, and other major comorbidities

At the same time, it clearly highlights the limit: effects are moderate, gradual and in no way comparable to drugs that directly and long-term activate GLP-1 receptors [1][3][5]

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