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S-glutathionylation has been shown to regulate the activity of numerous transcription factors and signaling proteins, including those in the NF-B, AP-1, and MAP kinase pathways (e.g., JNK, p38)

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By reinforcing this barrier, it prevents the translocation of luminal antigens and endotoxins into the bloodstream, a key pathological driver in many chronic inflammatory and autoimmune conditions

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BPC-157 remains investigational with limited human clinical data